Does Sudden Unexplained Nocturnal Death Syndrome Remain the Autopsy-Negative Disorder: A Gross, Microscopic, and Molecular Autopsy Investigation in Southern China von Zhang, Liyong, MD, Tester, David J., MS, Lang, Di, PhD, Chen, Yili, MD, PhD, Zheng, Jinxiang, MD, PhD, Gao, Rui, MS, Corliss, Robert F., MD, Tang, Shuangbo, MD, PhD, Kyle, John W., PhD, Liu, Chao, MS, Ackerman, Michael J., MD, PhD, Makielski, Jonathan C., MD, Cheng, Jianding, MD, PhD

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Channelopathies from mutations in the cardiac sodium channel protein complex von Adsit, Graham S, Vaidyanathan, Ravi, Galler, Carla M, Kyle, John W, Makielski, Jonathan C

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Molecular identity of the late sodium current in adult dog cardiomyocytes identified by Nav1.5 antisense inhibition von Maltsev, Victor A, Kyle, John W, Mishra, Sudhish, Undrovinas, Abertas

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Mechanisms of Sudden Cardiac Death: Oxidants and Metabolism von Yang, Kai-Chien, Kyle, John W, Makielski, Jonathan C, Dudley, Samuel C

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An East Asian Common Variant Vinculin P.Asp841His Was Associated With Sudden Unexplained Nocturnal Death Syndrome in the Chinese Han Population von Cheng, Jianding, Kyle, John W, Lang, Di, Wiedmeyer, Brandi, Guo, Jian, Yin, Kun, Huang, Lei, Vaidyanathan, Ravi, Su, Terry, Makielski, Jonathan C

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Late Na+ current produced by human cardiac Na+ channel isoform Nav1.5 is modulated by its β1 subunit von Maltsev, Victor A., Kyle, John W., Undrovinas, Albertas

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Transgenic overexpression of the SUR2A-55 splice variant in mouse heart reduces infract size and promotes protective mitochondrial function von Ramratnam, Mohun, Kenny, Barrett, Kyle, John W., Wiedmeyer, Brandi, Hacker, Timothy A., Barefield, David Y., McNally, Elizabeth M., Makielski, Jonathan C.

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Gating of the Late Na+ Channel in Normal and Failing Human Myocardium von Undrovinas, Albertas I., Maltsev, Victor A., Kyle, John W., Silverman, Norman, Sabbah, Hani N.

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Vinculin variant M94I identified in sudden unexplained nocturnal death syndrome decreases cardiac sodium current von Cheng, Jianding, Kyle, John W., Wiedmeyer, Brandi, Lang, Di, Vaidyanathan, Ravi, Makielski, Jonathan C.

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Toxin-Resistant Sodium Channels: Parallel Adaptive Evolution across a Complete Gene Family von Jost, Manda Clair, Hillis, David M., Lu, Ying, Kyle, John W., Fozzard, Harry A., Zakon, Harold H.

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Late INa in the Heart: Physiology, Pathology, and Pathways von Makielski, Jonathan C, Kyle, John W

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Structural organization of intercellular channels II. Amino terminal domain of the connexins: sequence, functional roles, and structure von Beyer, Eric C., Lipkind, Gregory M., Kyle, John W., Berthoud, Viviana M.

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An MIP/AQP0 mutation with impaired trafficking and function underlies an autosomal dominant congenital lamellar cataract von Senthil Kumar, G., Kyle, John W., Minogue, Peter J., Dinesh Kumar, K., Vasantha, K., Berthoud, Viviana M., Beyer, Eric C., Santhiya, S.T.

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A profile of alternative RNA splicing and transcript variation of CACNA1H, a human T-channel gene candidate for idiopathic generalized epilepsies von Zhong, Xiaoli, Liu, Jinrong R., Kyle, John W., Hanck, Dorothy A., Agnew, William S.

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The role of the putative inactivation lid in sodium channel gating current immobilization von Sheets, M F, Kyle, J W, Hanck, D A

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intact connexin N-terminus is required for function but not gap junction formation von Kyle, John W, Minogue, Peter J, Thomas, Bettina C, Domowicz, Denise A. Lopez, Berthoud, Viviana M, Hanck, Dorothy A, Beyer, Eric C

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The N Terminus of Connexin37 Contains an α-Helix That Is Required for Channel Function von Kyle, John W., Berthoud, Viviana M., Kurutz, Josh, Minogue, Peter J., Greenspan, Michael, Hanck, Dorothy A., Beyer, Eric C.

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The Na Channel Voltage Sensor Associated with Inactivation Is Localized to the External Charged Residues of Domain IV, S4 von Sheets, Michael F., Kyle, John W., Kallen, Roland G., Hanck, Dorothy A.

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Flecainide sensitivity of a Na channel long QT mutation shows an open-channel blocking mechanism for use-dependent block von Zhu, Yujie, Kyle, John W, Lee, Peter J

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Physiological Control of Smooth Muscle-specific Gene Expression through Regulated Nuclear Translocation of Serum Response Factor von Camoretti-Mercado, Blanca, Liu, Hong-W., Halayko, Andrew J., Forsythe, Sean M., Kyle, John W., Li, Bei, Fu, Yiping, McConville, John, Kogut, Paul, Vieira, Joaquim E., Patel, Nina M., Hershenson, Marc B., Fuchs, Elaine, Sinha, Satrajit, Miano, Joseph M., Parmacek, Michael S., Burkhardt, Janis K., Solway, Julian

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