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    Loss-of-function mutations in SLC30A8 protect against type 2 diabetes von Flannick, Jason, Thorleifsson, Gudmar, Beer, Nicola L, Jacobs, Suzanne B R, Grarup, Niels, Burtt, Noël P, Mahajan, Anubha, Fuchsberger, Christian, Atzmon, Gil, Benediktsson, Rafn, Blangero, John, Bowden, Don W, Brandslund, Ivan, Brosnan, Julia, Burslem, Frank, Chambers, John, Cho, Yoon Shin, Christensen, Cramer, Douglas, Desirée A, Duggirala, Ravindranath, Dymek, Zachary, Farjoun, Yossi, Fennell, Timothy, Fontanillas, Pierre, Forsén, Tom, Gabriel, Stacey, Glaser, Benjamin, Gudbjartsson, Daniel F, Hanis, Craig, Hansen, Torben, Hreidarsson, Astradur B, Hveem, Kristian, Ingelsson, Erik, Isomaa, Bo, Johansson, Stefan, Jørgensen, Torben, Jørgensen, Marit Eika, Kathiresan, Sekar, Kong, Augustine, Kooner, Jaspal, Kravic, Jasmina, Laakso, Markku, Lee, Jong-Young, Lind, Lars, Lindgren, Cecilia M, Linneberg, Allan, Masson, Gisli, Meitinger, Thomas, Mohlke, Karen L, Molven, Anders, Morris, Andrew P, Potluri, Shobha, Rauramaa, Rainer, Ribel-Madsen, Rasmus, Richard, Ann-Marie, Rolph, Tim, Salomaa, Veikko, Segrè, Ayellet V, Skärstrand, Hanna, Steinthorsdottir, Valgerdur, Stringham, Heather M, Sulem, Patrick, Tai, E Shyong, Teo, Yik Ying, Teslovich, Tanya, Thorsteinsdottir, Unnur, Trimmer, Jeff K, Tuomi, Tiinamaija, Tuomilehto, Jaakko, Vaziri-Sani, Fariba, Voight, Benjamin F, Wilson, James G, Boehnke, Michael, McCarthy, Mark I, Njølstad, Pål R, Pedersen, Oluf, Groop, Leif, Cox, David R, Stefansson, Kari, Altshuler, David

    Veröffentlicht in Nature genetics

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