An early endosome regulator, Rab5b, is an LRRK2 kinase substrate von Yun, Hye Jin, Kim, Hyejung, Ga, Inhwa, Oh, Hakjin, Ho, Dong Hwan, Kim, Jiyoung, Seo, Hyemyung, Son, Ilhong, Seol, Wongi

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Nuclear α-Synuclein-Derived Cytotoxic Effect via Altered Ribosomal RNA Processing in Primary Mouse Embryonic Fibroblasts von Ho, Dong Hwan, Kim, Hyejung, Nam, Daleum, Heo, Jinju, Son, Ilhong

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Expression of transduced nucleolin promotes the clearance of accumulated α-synuclein in rodent cells and animal model von Ho, Dong Hwan, Nam, Daleum, Jeong, Soyeon, Seo, Mi Kyoung, Park, Sung Woo, Seol, Wongi, Son, Ilhong

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Expression of G2019S LRRK2 in Rat Primary Astrocytes Mediates Neurotoxicity and Alters the Dopamine Synthesis Pathway in N27 Cells via Astrocytic Proinflammatory Cytokines and Neur... von Ho, Dong Hwan, Kim, Hyejung, Nam, Daleum, Seo, Mi Kyoung, Park, Sung Woo, Son, Ilhong

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Effects of Calcium Ion Dyshomeostasis and Calcium Ion-Induced Excitotoxicity in Parkinson’s Disease von Nam, Daleum, Kim, Hyejung, Sun Jung Han, Son, Ilhong, Ho, Dong Hwan

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LRRK2 Inhibition Mitigates the Neuroinflammation Caused by TLR2-Specific α-Synuclein and Alleviates Neuroinflammation-Derived Dopaminergic Neuronal Loss von Ho, Dong-Hwan, Nam, Daleum, Seo, Mikyoung, Park, Sung-Woo, Seol, Wongi, Son, Ilhong

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Therapeutic Effect of Padina arborescens Extract on a Cell System Model for Parkinson's Disease von Ho, Dong Hwan, Kim, Hyejung, Nam, Daleum, Seo, Mi Kyoung, Park, Sung Woo, Kim, Dong-Kyu, Son, Ilhong

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G2385R and I2020T Mutations Increase LRRK2 GTPase Activity von Seo, Hyemyung, Son, Ilhong, Joe, Eun-hye, Jang, Jihoon, Ho, Dong Hwan, Seol, Wongi

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G2019s LRRK2 promotes mitochondrial fission and increases TNFα-mediated neuroinflammation responses von Ho, Dong Hwan, Lee, Heajin, Son, Ilhong, Seol, Wongi

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Oxidized DJ-1 Levels in Urine Samples as a Putative Biomarker for Parkinson’s Disease von Ho, Dong Hwan, Seo, Hyemyung, Seol, Wongi, Lee, Sung Ik, Jeong, Soyeon, Jang, Jihoon, Son, Ilhong

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Effect of leucine-rich repeat kinase 2 (LRRK2) on protein synthesis von Kim, Hyejung, Son, Ilhong, Seol, Wongi

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Analysis of α-synuclein levels related to LRRK2 kinase activity: from substantia nigra to urine of patients with Parkinson's disease von Nam, Daleum, Kim, Ami, Han, Sun Jung, Lee, Sung-Ik, Park, Sung-Hye, Seol, Wongi, Son, Ilhong, Ho, Dong Hwan

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Upregulation of the p53-p21 pathway by G2019S LRRK2 contributes to the cellular senescence and accumulation of α-synuclein von Ho, Dong Hwan, Seol, Wongi, Son, Ilhong

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KCHO-1, a novel herbal anti-inflammatory compound, attenuates oxidative stress in an animal model of amyotrophic lateral sclerosis von Kook, Myung Geun, Choi, Soon Won, Seo, Yoojin, Kim, Dong Woung, Song, Bong Keun, Son, Ilhong, Kim, Sungchul, Kang, Kyung-Sun

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Mecasin treatment in patients with amyotrophic lateral sclerosis: study protocol for a randomized controlled trial von Kim, Sungha, Kim, Jae Kyoun, Son, Mi Ju, Kim, Dongwoung, Song, Bongkeun, Son, Ilhong, Kang, Hyung Won, Lee, Jongdeok, Kim, Sungchul

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Increase in anti-apoptotic molecules, nucleolin, and heat shock protein 70, against upregulated LRRK2 kinase activity von Jang, Jihoon, Oh, Hakjin, Nam, Daleum, Seol, Wongi, Seo, Mi Kyoung, Park, Sung Woo, Kim, Hyung Gun, Seo, Hyemyung, Son, Ilhong, Ho, Dong Hwan

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26-Week Repeated Dose Oral Toxicity Study of KCHO-1 in Sprague-Dawley Rats von Yang, Muhack, Lee, Seongjin, Wang, Tingting, Cha, Eunhye, Jang, Jongwon, Kim, Dongwoung, Song, Bong-Keun, Son, Ilhong, Kim, Joonyup, Kang, Hyung Won, Kim, Sungchul

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Correction to: Leucine-Rich Repeat Kinase 2 (LRRK2) phosphorylates p53 and induces p21WAF1/CIP1 expression von Ho, Dong Hwan, Kim, Hyejung, Kim, Jisun, Sim, Hyuna, Ahn, Hyunjun, Kim, Janghwan, Seo, Hyemyung, Chung, Kwang Chul, Park, Bum-Joon, Son, Ilhong, Seol, Wongi

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Characterization of Parkinson's disease-related pathogenic TMEM230 mutants von Nam, Daleum, Kim, Hyejung, Choi, Dong-Joo, Bae, Yun-Hee, Lee, Byoung Dae, Son, Ilhong, Seol, Wongi

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Dexamethasone induces the expression of LRRK2 and α-synuclein, two genes that when mutated cause Parkinson's disease in an autosomal dominant manner von Park, Ji-Min, Ho, Dong-Hwan, Yun, Hye Jin, Kim, Hye-Jung, Lee, Chan Hong, Park, Sung Woo, Kim, Young Hoon, Son, Ilhong, Seol, Wongi

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