PINK1-Parkin signaling in Parkinson’s disease: Lessons from Drosophila von Imai, Yuzuru

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Editorial: Molecular links between mitochondrial damage and human neurodegenerative disorders, volume II von Imai, Yuzuru

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Pathogenic LRRK2 negatively regulates microRNA-mediated translational repression von Gehrke, Stephan, Imai, Yuzuru, Sokol, Nicholas, Lu, Bingwei

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Editorial for the Special Issue "Animal Models of Parkinson's Disease and Related Disorders" von Imai, Yuzuru

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PINK1-mediated phosphorylation of the Parkin ubiquitin-like domain primes mitochondrial translocation of Parkin and regulates mitophagy von Shiba-Fukushima, Kahori, Imai, Yuzuru, Yoshida, Shigeharu, Ishihama, Yasushi, Kanao, Tomoko, Sato, Shigeto, Hattori, Nobutaka

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Twin CHCH Proteins, CHCHD2, and CHCHD10: Key Molecules of Parkinson's Disease, Amyotrophic Lateral Sclerosis, and Frontotemporal Dementia von Imai, Yuzuru, Meng, Hongrui, Shiba-Fukushima, Kahori, Hattori, Nobutaka

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Loss of Parkinson’s disease-associated protein CHCHD2 affects mitochondrial crista structure and destabilizes cytochrome c von Meng, Hongrui, Yamashita, Chikara, Shiba-Fukushima, Kahori, Inoshita, Tsuyoshi, Funayama, Manabu, Sato, Shigeto, Hatta, Tomohisa, Natsume, Tohru, Umitsu, Masataka, Takagi, Junichi, Imai, Yuzuru, Hattori, Nobutaka

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Legumain/asparaginyl endopeptidase-resistant tau fibril fold produces corticobasal degeneration-specific C-terminal tau fragment von Taniguchi, Daisuke, Shimonaka, Shotaro, Imtiaz, Ahmed, Elahi, Montasir, Hatano, Taku, Imai, Yuzuru, Hattori, Nobutaka

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Mitochondrial dynamics and mitophagy in Parkinson's disease: disordered cellular power plant becomes a big deal in a major movement disorder von Imai, Yuzuru, Lu, Bingwei

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Syntaxin 17 regulates the localization and function of PGAM5 in mitochondrial division and mitophagy von Sugo, Masashi, Kimura, Hana, Arasaki, Kohei, Amemiya, Toshiki, Hirota, Naohiko, Dohmae, Naoshi, Imai, Yuzuru, Inoshita, Tsuyoshi, Shiba‐Fukushima, Kahori, Hattori, Nobutaka, Cheng, Jinglei, Fujimoto, Toyoshi, Wakana, Yuichi, Inoue, Hiroki, Tagaya, Mitsuo

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High-fat diet–induced activation of SGK1 promotes Alzheimer’s disease–associated tau pathology von Elahi, Montasir, Motoi, Yumiko, Shimonaka, Shotaro, Ishida, Yoko, Hioki, Hiroyuki, Takanashi, Masashi, Ishiguro, Koichi, Imai, Yuzuru, Hattori, Nobutaka

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Experimental Models of Parkinson’s Disease von Imai, Yuzuru

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PINK1-mediated phosphorylation of Parkin boosts Parkin activity in Drosophila von Shiba-Fukushima, Kahori, Inoshita, Tsuyoshi, Hattori, Nobutaka, Imai, Yuzuru

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Parkinson's disease-associated kinase PINK1 regulates Miro protein level and axonal transport of mitochondria von Liu, Song, Sawada, Tomoyo, Lee, Seongsoo, Yu, Wendou, Silverio, George, Alapatt, Philomena, Millan, Ivan, Shen, Alice, Saxton, William, Kanao, Tomoko, Takahashi, Ryosuke, Hattori, Nobutaka, Imai, Yuzuru, Lu, Bingwei

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Phosphorylation of mitochondrial polyubiquitin by PINK1 promotes Parkin mitochondrial tethering von Shiba-Fukushima, Kahori, Arano, Taku, Matsumoto, Gen, Inoshita, Tsuyoshi, Yoshida, Shigeharu, Ishihama, Yasushi, Ryu, Kwon-Yul, Nukina, Nobuyuki, Hattori, Nobutaka, Imai, Yuzuru

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Phosphorylation of 4E-BP by LRRK2 affects the maintenance of dopaminergic neurons in Drosophila von Imai, Yuzuru, Gehrke, Stephan, Wang, Hua-Qin, Takahashi, Ryosuke, Hasegawa, Kazuko, Oota, Etsuro, Lu, Bingwei

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Is Glial Dysfunction the Key Pathogenesis of LRRK2 -Linked Parkinson's Disease? von Iseki, Tatou, Imai, Yuzuru, Hattori, Nobutaka

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Ubiquitination at the lysine 27 residue of the Parkin ubiquitin-like domain is suggestive of a new mechanism of Parkin activation von Liu, Jun-Yi, Inoshita, Tsuyoshi, Shiba-Fukushima, Kahori, Yoshida, Shigeharu, Ogata, Kosuke, Ishihama, Yasushi, Imai, Yuzuru, Hattori, Nobutaka

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Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused by inactivation of Drosophila Pink1 is rescued by Parkin von Yang, Y, Gehrke, S, Imai, Y, Huang, Z, Ouyang, Y, Wang, J.W, Yang, L, Beal, M.F, Vogel, H, Lu, B

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Neurodegeneration-associated mitochondrial proteins, CHCHD2 and CHCHD10–what distinguishes the two? von Ikeda, Aya, Imai, Yuzuru, Hattori, Nobutaka

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