Decoding ALS: from genes to mechanism von Taylor, J. Paul, Brown, Robert H., Cleveland, Don W.

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Emerging mechanisms of molecular pathology in ALS von Peters, Owen M, Ghasemi, Mehdi, Brown, Jr, Robert H

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Intrinsic membrane hyperexcitability of amyotrophic lateral sclerosis patient-derived motor neurons von Wainger, Brian J, Kiskinis, Evangelos, Mellin, Cassidy, Wiskow, Ole, Han, Steve S W, Sandoe, Jackson, Perez, Numa P, Williams, Luis A, Lee, Seungkyu, Boulting, Gabriella, Berry, James D, Brown, Jr, Robert H, Cudkowicz, Merit E, Bean, Bruce P, Eggan, Kevin, Woolf, Clifford J

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Amyotrophic lateral sclerosis: Problems and prospects von Sreedharan, Jemeen, Brown Jr, Robert H.

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Genetics of Amyotrophic Lateral Sclerosis von Ghasemi, Mehdi, Brown, Jr, Robert H

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Suppression of mutant C9orf72 expression by a potent mixed backbone antisense oligonucleotide von Tran, Hélène, Moazami, Michael P., Yang, Huiya, McKenna-Yasek, Diane, Douthwright, Catherine L., Pinto, Courtney, Metterville, Jake, Shin, Minwook, Sanil, Nitasha, Dooley, Craig, Puri, Ajit, Weiss, Alexandra, Wightman, Nicholas, Gray-Edwards, Heather, Marosfoi, Miklos, King, Robert M., Kenderdine, Thomas, Fabris, Daniele, Bowser, Robert, Watts, Jonathan K., Brown, Robert H.

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Molecular biology of amyotrophic lateral sclerosis: insights from genetics von Pasinelli, Piera, Brown, Robert H.

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Human C9ORF72 Hexanucleotide Expansion Reproduces RNA Foci and Dipeptide Repeat Proteins but Not Neurodegeneration in BAC Transgenic Mice von Peters, Owen M., Cabrera, Gabriela Toro, Tran, Helene, Gendron, Tania F., McKeon, Jeanne E., Metterville, Jake, Weiss, Alexandra, Wightman, Nicholas, Salameh, Johnny, Kim, Juhyun, Sun, Huaming, Boylan, Kevin B., Dickson, Dennis, Kennedy, Zachary, Lin, Ziqiang, Zhang, Yong-Jie, Daughrity, Lillian, Jung, Chris, Gao, Fen-Biao, Sapp, Peter C., Horvitz, H. Robert, Bosco, Daryl A., Brown, Solange P., de Jong, Pieter, Petrucelli, Leonard, Mueller, Christian, Brown, Robert H.

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Gene therapy for neurologic disorders von Eichler, Florian S., Brown, Robert H.

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Wild-type and mutant SOD1 share an aberrant conformation and a common pathogenic pathway in ALS von Bosco, Daryl A, Morfini, Gerardo, Karabacak, N Murat, Song, Yuyu, Gros-Louis, Francois, Pasinelli, Piera, Goolsby, Holly, Fontaine, Benjamin A, Lemay, Nathan, McKenna-Yasek, Diane, Frosch, Matthew P, Agar, Jeffrey N, Julien, Jean-Pierre, Brady, Scott T, Brown, Robert H

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Silencing strategies for therapy of SOD1-mediated ALS von van Zundert, Brigitte, Brown, Robert H.

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Adeno-associated virus-delivered artificial microRNA extends survival and delays paralysis in an amyotrophic lateral sclerosis mouse model von Stoica, Lorelei, Todeasa, Sophia H., Cabrera, Gabriela Toro, Salameh, Johnny S., ElMallah, Mai K., Mueller, Christian, Brown Jr, Robert H., Sena-Esteves, Miguel

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EPHA4 is a disease modifier of amyotrophic lateral sclerosis in animal models and in humans von Van Hoecke, Annelies, Schoonaert, Lies, Lemmens, Robin, Timmers, Mieke, Staats, Kim A, Laird, Angela S, Peeters, Elke, Philips, Thomas, Goris, An, Dubois, Bénédicte, Andersen, Peter M, Al-Chalabi, Ammar, Thijs, Vincent, Turnley, Ann M, van Vught, Paul W, Veldink, Jan H, Hardiman, Orla, Van Den Bosch, Ludo, Gonzalez-Perez, Paloma, Van Damme, Philip, Brown, Robert H, van den Berg, Leonard H, Robberecht, Wim

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TDP-43 gains function due to perturbed autoregulation in a Tardbp knock-in mouse model of ALS-FTD von White, Matthew A., Kim, Eosu, Duffy, Amanda, Adalbert, Robert, Phillips, Benjamin U., Peters, Owen M., Stephenson, Jodie, Yang, Sujeong, Massenzio, Francesca, Lin, Ziqiang, Andrews, Simon, Segonds-Pichon, Anne, Metterville, Jake, Saksida, Lisa M., Mead, Richard, Ribchester, Richard R, Barhomi, Youssef, Serre, Thomas, Coleman, Michael P., Fallon, Justin R., Bussey, Timothy J., Brown, Robert H., Sreedharan, Jemeen

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Hereditary Sensory Neuropathy Type 1 Is Caused by the Accumulation of Two Neurotoxic Sphingolipids von Penno, Anke, Reilly, Mary M., Houlden, Henry, Laurá, Matilde, Rentsch, Katharina, Niederkofler, Vera, Stoeckli, Esther T., Nicholson, Garth, Eichler, Florian, Brown, Robert H., von Eckardstein, Arnold, Hornemann, Thorsten

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Axonal Transport Defects in Neurodegenerative Diseases von Morfini, Gerardo A, Burns, Matthew, Binder, Lester I, Kanaan, Nicholas M, LaPointe, Nichole, Bosco, Daryl A, Brown, Robert H., Jr, Brown, Hannah, Tiwari, Ashutosh, Hayward, Lawrence, Edgar, Julia, Nave, Klaus-Armin, Garberrn, James, Atagi, Yuka, Song, Yuyu, Pigino, Gustavo, Brady, Scott T

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dSarm/Sarm1 Is Required for Activation of an Injury-Induced Axon Death Pathway von Osterloh, Jeannette M., Yang, Jing, Rooney, Timothy M., Fox, A. Nicole, Adalbert, Robert, Powell, Eric H., Sheehan, Amy E., Avery, Michelle A., Hackett, Rachel, Logan, Mary A., MacDonald, Jennifer M., Ziegenfuss, Jennifer S., Milde, Stefan, Hou, Ying-Ju, Nathan, Carl, Ding, Aihao, Brown, Robert H., Conforti, Laura, Coleman, Michael, Tessier-Lavigne, Marc, Züchner, Stephan, Freeman, Marc R.

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A randomized placebo‐controlled phase 3 study of mesenchymal stem cells induced to secrete high levels of neurotrophic factors in amyotrophic lateral sclerosis von Cudkowicz, Merit E., Lindborg, Stacy R., Goyal, Namita A., Miller, Robert G., Burford, Matthew J., Berry, James D., Nicholson, Katharine A., Mozaffar, Tahseen, Katz, Jonathan S., Jenkins, Liberty J., Baloh, Robert H., Lewis, Richard A., Staff, Nathan P., Owegi, Margaret A., Berry, Donald A., Gothelf, Yael, Levy, Yossef S., Aricha, Revital, Kern, Ralph Z., Windebank, Anthony J., Brown, Robert H.

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Partial loss of TDP-43 function causes phenotypes of amyotrophic lateral sclerosis von Yang, Chunxing, Wang, Hongyan, Qiao, Tao, Yang, Bin, Aliaga, Leonardo, Qiu, Linghua, Tan, Weijia, Salameh, Johnny, McKenna-Yasek, Diane M., Smith, Thomas, Peng, Lingtao, Moore, Melissa J., Brown, Robert H., Cai, Huaibin, Xu, Zuoshang

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T lymphocytes potentiate endogenous neuroprotective inflammation in a mouse model of ALS von Chiu, Isaac M, Chen, Adam, Zheng, Yi, Kosaras, Bela, Tsiftsoglou, Stefanos A, Vartanian, Timothy K, Brown, Robert H. Jr, Carroll, Michael C

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