Historical Perspective of the Characterization of Conotoxins Targeting Voltage-Gated Sodium Channels von Groome, James R

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Roles for Countercharge in the Voltage Sensor Domain of Ion Channels von Groome, James R., Bayless-Edwards, Landon

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Nav1.4 mutations cause hypokalaemic periodic paralysis by disrupting IIIS4 movement during recovery von GROOME, James R, LEHMANN-HORN, Frank, CHUNXIANG FAN, WOLF, Markus, WINSTON, Vern, MERLINI, Luciano, JURKAT-ROTT, Karin

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Gating defects in K1126I, a putative hypokalemic periodic paralysis mutation in hNav1.4 von Groome, James R.

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S1-S3 counter charges in the voltage sensor module of a mammalian sodium channel regulate fast inactivation von Groome, James R, Winston, Vern

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NaV1.4 DI-S4 periodic paralysis mutation R222W enhances inactivation and promotes leak current to attenuate action potentials and depolarize muscle fibers von Bayless-Edwards, Landon, Winston, Vern, Lehmann-Horn, Frank, Arinze, Paula, Groome, James R., Jurkat-Rott, Karin

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Domain III S4 in closed-state fast inactivation: Insights from a periodic paralysis mutation von Groome, James R, Jurkat-Rott, Karin, Lehmann-Horn, Frank

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The Voltage Sensor Module in Sodium Channels von Groome, James R.

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Correction: S1-S3 counter charges in the voltage sensor module of a mammalian sodium channel regulate fast inactivation von Groome, James R, Winston, Vern

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Transient compartment-like syndrome and normokalaemic periodic paralysis due to a Ca(v)1.1 mutation von Fan, Chunxiang, Lehmann-Horn, Frank, Weber, Marc-André, Bednarz, Marcin, Groome, James R, Jonsson, Malin K B, Jurkat-Rott, Karin

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Transient compartment-like syndrome and normokalaemic periodic paralysis due to a Cav1.1 mutation von CHUNXIANG FAN, LEHMANN-HORN, Frank, WEBER, Marc-André, BEDNARZ, Marcin, GROOME, James R, JONSSON, Malin K. B, JURKAT-ROTT, Karin

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Ribbon α-Conotoxin KTM Exhibits Potent Inhibition of Nicotinic Acetylcholine Receptors von Marquart, Leanna A, Turner, Matthew W, Warner, Lisa R, King, Matthew D, Groome, James R, McDougal, Owen M

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Temperature-sensitive defects in paramyotonia congenita mutants R1448C and T1313M von Dice, Margaret S., Abbruzzese, Jennifer L., Wheeler, James T., Groome, James R., Fujimoto, Esther, Ruben, Peter C.

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Open- and closed-state fast inactivation in sodium channels: Differential effects of a site-3 anemone toxin von Groome, James, Lehmann-Horn, Frank, Holzherr, Boris

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Characterizing gating defects of SCN2A countercharge mutations in epilepsy syndromes von Groome, James R., Brockway, Sarah N.

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A negative charge in transmembrane segment 1 of domain II of the cockroach sodium channel is critical for channel gating and action of pyrethroid insecticides von Du, Yuzhe, Song, Weizhong, Groome, James R., Nomura, Yoshiko, Luo, Ningguang, Dong, Ke

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Differential effects of homologous S4 mutations in human skeletal muscle sodium channels on deactivation gating from open and inactivated states von James R Groome, Esther Fujimoto, Alfred L George, Jr, Peter C Ruben

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Role for Fast Inactivation in Domain I of Voltage Gated Sodium Channels von Groome, James R., Camp, Ryann

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Domain I Countercharges Limit Slow Inactivation in hNav1.4 Sodium Channels von Groome, James R., Wheeler, Andromeda, Camp, Ryann

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Voltage Sensor Domains and Closed-State Inactivation in Sodium Channels von Groome, James R.

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