Goodbye to the bioassay von Goodman, Jay I

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The Constitutive Active/Androstane Receptor Facilitates Unique Phenobarbital-Induced Expression Changes of Genes Involved in Key Pathways in Precancerous Liver and Liver Tumors von Phillips, Jennifer M., Burgoon, Lyle D., Goodman, Jay I.

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In Memoriam: Jerry B. Hook (1938 –2021) von Goodman, Jay I, Kaminski, Norbert E

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Altered DNA methylation: a secondary mechanism involved in carcinogenesis von Goodman, Jay I, Watson, Rebecca E

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The FEMA GRAS assessment of cinnamyl derivatives used as flavor ingredients von Adams, Timothy B, Cohen, Samuel M, Doull, John, Feron, Victor J, Goodman, Jay I, Marnett, Lawrence J, Munro, Ian C, Portoghese, Philip S, Smith, Robert L, Waddell, William J, Wagner, Bernard M

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Correction: Goodbye to the bioassay von Goodman, Jay I

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Categorizing the characteristics of human carcinogens: a need for specificity von Smith, Carr J., Perfetti, Thomas A., Hayes, A. Wallace, Berry, Sir Colin, Trosko, James E., King, Judy A., Goodman, Jay I., Begley, C. Glenn, Dayan, Anthony

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Incorporation of an epigenetic evaluation into safety assessment: What we first need to know von Goodman, Jay I.

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Computational modeling identifies key gene regulatory interactions underlying phenobarbital-mediated tumor promotion von Luisier, Raphaëlle, Unterberger, Elif B, Goodman, Jay I, Schwarz, Michael, Moggs, Jonathan, Terranova, Rémi, van Nimwegen, Erik

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Phenobarbital and propiconazole toxicogenomic profiles in mice show major similarities consistent with the key role that constitutive androstane receptor (CAR) activation plays in... von Currie, Richard A, Peffer, Richard C, Goetz, Amber K, Omiecinski, Curtis J, Goodman, Jay I

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Response to Druwe and Burgoon, 2016 Letter to the Editor in Archives of Toxicology von Cohen, Samuel M., Goodman, Jay I., Klaunig, James E., Arnold, Lora L.

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Response to Druwe and Burgoon: Re: Druwe, I.L. and Burgoon, L.: revisiting Cohen et al. 2015, Cohen et al. 2014 and Waalkes et al. 2014: a bayesian re-analysis of tumor incidences von Cohen, Samuel M., Arnold, Lora L., Klaunig, James E., Goodman, Jay I.

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Multiple Genes Exhibit Phenobarbital-Induced Constitutive Active/Androstane Receptor–Mediated DNA Methylation Changes during Liver Tumorigenesis and in Liver Tumors von Phillips, Jennifer M., Goodman, Jay I.

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Re: Waalkes et al.: Lung tumors in mice induced by “whole-life” inorganic arsenic exposure at human-relevant doses, Arch Toxicol, 2014 von Cohen, Samuel M., Arnold, Lora L., Klaunig, James E., Goodman, Jay I.

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Identification of Genes that May Play Critical Roles in Phenobarbital (PB)-Induced Liver Tumorigenesis due to Altered DNA Methylation von Phillips, Jennifer M., Goodman, Jay I.

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Phenobarbital Elicits Unique, Early Changes in the Expression of Hepatic Genes that Affect Critical Pathways in Tumor-Prone B6C3F1 Mice von Phillips, Jennifer M., Burgoon, Lyle D., Goodman, Jay I.

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Changes in DNA methylation and gene expression during 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced suppression of the lipopolysaccharide-stimulated IgM response in splenocytes von McClure, Emily A, North, Colin M, Kaminski, Norbert E, Goodman, Jay I

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What do we need to know prior to thinking about incorporating an epigenetic evaluation into safety assessments? von Goodman, Jay I, Augustine, Karen A, Cunnningham, Michael L, Dixon, Darlene, Dragan, Yvonne P, Falls, James Greg, Rasoulpour, Reza J, Sills, Robert C, Storer, Richard D, Wolf, Douglas C, Pettit, Syril D

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Phenobarbital Induces Progressive Patterns of GC-Rich and Gene-Specific Altered DNA Methylation in the Liver of Tumor-Prone B6C3F1 Mice von Bachman, Ammie N., Phillips, Jennifer M., Goodman, Jay I.

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Obfuscating transparency? von Aschner, Michael, Autrup, Herman, Berry, Colin L., Boobis, Alan R., Cohen, Samuel M., Dekant, Wolfgang, Galli, Corrado L., Goodman, Jay I., Gori, Gio B., Greim, Helmut A., Kaminski, Norbert E., Klaassen, Curtis D., Klaunig, James E., Lotti, Marcello, Marquardt, Hans WJ, Moretto, Angelo, Pelkonen, Olavi, Sipes, I. Glenn, Wallace, Kendall B., Yamazaki, Hiroshi

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