Chk2 is required for optimal mitotic delay in response to irradiation-induced DNA damage incurred in G2 phase von Rainey, M D, Black, E J, Zachos, G, Gillespie, D A F

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Chk1-dependent slowing of S-phase progression protects DT40 B-lymphoma cells against killing by the nucleoside analogue 5-fluorouracil von Robinson, H M R, Jones, R, Walker, M, Zachos, G, Brown, R, Cassidy, J, Gillespie, D A F

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v-Jun stimulates both cdk2 kinase activity and G1 S progression via transcriptional repression of p21 CIP1 von MACLAREN, A, CLARK, W, BLACK, E. J, GREGORY, D, FUJII, H, GILLESPIE, D. A. F

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v-Jun sensitizes cells to apoptosis by a mechanism involving mitochondrial cytochrome C release von MACLAREN, A, CLARK, W, GILLESPIE, D. A. F

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Viral mutations enhance the Max binding properties of the vMyc b-HLH-LZ domain von Crouch, D. H., Fisher, F., La Rocca, S. A., Goding, C. R., Gillespie, D. A. F.

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v-Jun Overrides the Mitogen Dependence of S-Phase Entry by Deregulating Retinoblastoma Protein Phosphorylation and E2F-Pocket Protein Interactions as a Consequence of Enhanced Cycl... von Clark, W., Black, E. J., MacLaren, A., Kruse, U., LaThangue, N., Vogt, P. K., Gillespie, D. A. F.

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Chk2 is required for optimal mitotic delay in response to irradiation-induced DNA damage incurred in G phase

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Chk2 is required for optimal mitotic delay in response to irradiation- induced DNA damage incurred in G sub(2) phase von Rainey, M D, Black, E J, Zachos, G, Gillespie, D A F

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Chkl-dependent slowing of S-phase progression protects DT40 B-lymphoma cells against killing by the nucleoside analogue 5-fluorouracil von ROBINSON, H. M. R, JONES, R, WALKER, M, ZACHOS, G, BROWN, R, CASSIDY, J, GILLESPIE, D. A. F

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Transcription activation by Myc and Max: flanking sequences target activation to a subset of CACGTG motifs in vivo von Fisher, F., Crouch, D.H., Jayaraman, P.S., Clark, W., Gillespie, D.A., Goding, C.R.

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Transformation by the fos or jun oncogene does not increase AP-1 DNA-binding activity von HAWKER, K. L, PINTZAS, A, HENNIGAN, R. F, GILLESPIE, D. A. F, OZANNE, B. W

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Detection of a Myc-associated protein by chemical cross-linking von GILLESPIE, D. A. F, EISENMAN, R. N

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VMyc can sustain longer deletions in the LZ than cMyc before loss of biological activity () Cell morphology of CEF expressing cMyc, vMyc and their respective LZ mutants are shown von D. H. Crouch, F. Fisher, S. A. La Rocca, C. R. Goding, D. A. F. Gillespie

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Sequence alignment of the LZ domains of different transcription factors von D. H. Crouch, F. Fisher, S. A. La Rocca, C. R. Goding, D. A. F. Gillespie

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IL mutation in cMycÎ 10 partially restores interaction with Max/Max9 and von D. H. Crouch, F. Fisher, S. A. La Rocca, C. R. Goding, D. A. F. Gillespie

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Complex formation between Max/Max9 and the LZ mutants of v- and c-Myc in yeast von D. H. Crouch, F. Fisher, S. A. La Rocca, C. R. Goding, D. A. F. Gillespie

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Complex formation between Max/Max9 and the LZ mutants of v- and c-Myc von D. H. Crouch, F. Fisher, S. A. La Rocca, C. R. Goding, D. A. F. Gillespie

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LZ mutants of MC29 vMyc and avian cMyc von D. H. Crouch, F. Fisher, S. A. La Rocca, C. R. Goding, D. A. F. Gillespie

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Expression of integrated Rous sarcoma viruses: DNA rearrangements 5′ to the provirus are common in transformed rat cells but not seen in infected but untransformed cells von Green, A.R., Searle, S., Gillespie, D.A., Bissell, M., Wyke, J.A.

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Transformation by v-Jun prevents cell cycle exit and promotes apoptosis in the absence of serum growth factors von CLARK, W, GILLESPIE, D. A. F

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