New Therapeutic Strategies for Osteoarthritis by Targeting Sialic Acid Receptors von Carpintero-Fernandez, Paula, Varela-Eirin, Marta, Lacetera, Alessandra, Gago-Fuentes, Raquel, Fonseca, Eduardo, Martin-Santamaria, Sonsoles, Mayan, Maria D

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Normal development of mice lacking PAXX, the paralogue of XRCC4 and XLF von Gago‐Fuentes, Raquel, Xing, Mengtan, Sæterstad, Siri, Sarno, Antonio, Dewan, Alisa, Beck, Carole, Bradamante, Stefano, Bjørås, Magnar, Oksenych, Valentyn

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Robust DNA repair in PAXX‐deficient mammalian cells von Dewan, Alisa, Xing, Mengtan, Lundbæk, Marie Benner, Gago‐Fuentes, Raquel, Beck, Carole, Aas, Per Arne, Liabakk, Nina‐Beate, Sæterstad, Siri, Chau, Khac Thanh Phong, Kavli, Bodil Merete, Oksenych, Valentyn

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Non-Homologous End Joining Factors XLF, PAXX and DNA-PKcs Maintain the Neural Stem and Progenitor Cell Population von Gago-Fuentes, Raquel, Oksenych, Valentyn

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OP0308 CELL –To-Cell Communication VIA GAP Junctions Between Cartilage, Synovial Membrane and Subchondral Bone: Implications for Joint Homeostasis von Carpintero-Fernández, P., Fernández-Puente, P., Raquel Gago-Fuentes, R., Blanco, F.J., Brink, P.R., Mayán, M.D.

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Normal development of mice lacking PAXX , the paralogue of XRCC 4 and XLF von Gago‐Fuentes, Raquel, Xing, Mengtan, Sæterstad, Siri, Sarno, Antonio, Dewan, Alisa, Beck, Carole, Bradamante, Stefano, Bjørås, Magnar, Oksenych, Valentyn

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Synthetic lethality between DNA repair factors Xlf and Paxx is rescued by inactivation of Trp53 von Castañeda-Zegarra, Sergio, Xing, Mengtan, Gago-Fuentes, Raquel, Sæterstad, Siri, Oksenych, Valentyn

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Non-Homologous End Joining Factors XLF, PAXX and DNA-PKcs Maintain the Neural Stem and Progenitor Cell Population von Gago-Fuentes, Raquel, Oksenych, Valentyn

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Generation of a Mouse Model Lacking the Non-Homologous End-Joining Factor Mri/Cyren von Castañeda-Zegarra, Sergio, Huse, Camilla, Røsand, Øystein, Sarno, Antonio, Xing, Mengtan, Gago-Fuentes, Raquel, Zhang, Qindong, Alirezaylavasani, Amin, Werner, Julia, Ji, Ping, Liabakk, Nina-Beate, Wang, Wei, Bjørås, Magnar, Oksenych, Valentyn

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Articular chondrocyte network mediated by gap junctions: role in metabolic cartilage homeostasis von Mayan, Maria D, Gago-Fuentes, Raquel, Carpintero-Fernandez, Paula, Fernandez-Puente, Patricia, Filgueira-Fernandez, Purificacion, Goyanes, Noa, Valiunas, Virginijus, Brink, Peter R, Goldberg, Gary S, Blanco, Francisco J

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Intercellular communication via gap junction channels between chondrocytes and bone cells von Carpintero-Fernandez, Paula, Gago-Fuentes, Raquel, Wang, Hong Z., Fonseca, Eduardo, Caeiro, José R., Valiunas, Virginijus, Brink, Peter R., Mayan, Maria D.

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Human articular chondrocytes express multiple gap junction proteins: differential expression of connexins in normal and osteoarthritic cartilage von Mayan, Maria D, Carpintero-Fernandez, Paula, Gago-Fuentes, Raquel, Martinez-de-Ilarduya, Oskar, Wang, Hong-Zhang, Valiunas, Virginijus, Brink, Peter, Blanco, Francisco J

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Proteomic Analysis of Connexin 43 Reveals Novel Interactors Related to Osteoarthritis von Gago-Fuentes, Raquel, Fernández-Puente, Patricia, Megias, Diego, Carpintero-Fernández, Paula, Mateos, Jesus, Acea, Benigno, Fonseca, Eduardo, Blanco, Francisco Javier, Mayan, Maria Dolores

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Wounding induces local resistance but systemic susceptibility to Botrytis cinerea in pepper plants von García, Tania, Gutiérrez, Jorge, Veloso, Javier, Gago-Fuentes, Raquel, Díaz, José

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The C-terminal domain of connexin43 modulates cartilage structure via chondrocyte phenotypic changes von Gago-Fuentes, Raquel, Bechberger, John F, Varela-Eirin, Marta, Varela-Vazquez, Adrian, Acea, Benigno, Fonseca, Eduardo, Naus, Christian C, Mayan, Maria D

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Human Articular Chondrocytes Express Multiple Gap Junction Proteins von Mayan, Maria D, Carpintero-Fernandez, Paula, Gago-Fuentes, Raquel, Martinez-de-Ilarduya, Oskar, Wang, Hong-Zhang, Valiunas, Virginijus, Brink, Peter, Blanco, Francisco J

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Non-Homologous End Joining Factors XLF, PAXX and DNA-PKcs Maintain the Neural Stem and Progenitor Cell Population von Gago-Fuentes, Raquel, Oksenych, Valentyn

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Synthetic lethality between DNA repair factors Xlf and Paxx is rescued by inactivation of Trp53 von Castañeda-Zegarra, Sergio, Xing, Mengtan, Gago-Fuentes, Raquel, Sæterstad, Siri, Oksenych, Valentyn

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Normal development of mice lacking PAXX, the paralogue of XRCC4 and XLF von Gago-Fuentes, Raquel, Xing, Mengtan, Sæterstad, Siri, Sarno, Antonio, Dewan, Alisa Elinsdatter, Beck, Carole, Bradamante, Stefano, Bjørås, Magnar, Oksenych, Valentyn

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Robust DNA repair in PAXX-deficient mammalian cells von Dewan, Alisa Elinsdatter, Xing, Mengtan, Lundbæk, Marie Benner, Gago-Fuentes, Raquel, Beck, Carole, Aas, Per Arne, Liabakk, Nina-Beate, Sæterstad, Siri, Khac Thanh Phong, Chau, Kavli, Bodil Merete, Oksenych, Valentyn

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