The scid mutation in mice causes a general defect in DNA repair von Fulop, G. M, Phillips, R. A

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The scid defect affects the final step of the immunoglobulin VDJ recombinase mechanism von Malynn, Barbara A., Blackwell, T.Keith, Fulop, Gabrielle M., Rathbun, Gary A., Furley, Andrew J.W., Ferrier, Pierre, Heinke, L.Bruce, Phillips, Robert A., Yancopoulos, George D., Alt, Frederick W.

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Isolation of scid pre‐B cells that rearrange kappa light chain genes: formation of normal signal and abnormal coding joins von Blackwell, T. K., Malynn, B. A., Pollock, R. R., Ferrier, P., Covey, L. R., Fulop, G. M., Phillips, R. A., Yancopoulos, G. D., Alt, F. W.

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Full reconstitution of the immune deficiency in scid mice with normal stem cells requires low-dose irradiation of the recipients von Fulop, GM, Phillips, RA

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Use of scid Mice to Identify and Quantitate Lymphoid-Restricted Stem Cells in Long-Term Bone Marrow Cultures von Fulop, G.M., Phillips, R.A.

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Use of scid mice to identify and quantitate lymphoid-restricted stem cells in long-term bone marrow cultures von Fulop, GM, Phillips, RA

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An expanded population of natural killer cells in mice with severe combined immunodeficiency (SCID) lack rearrangement and expression of T cell receptor genes von LAUZON, R. J, SIMINOVITCH, K. A, FULOP, G. M, PHILLIPS, R. A, RODER, J. C

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Early B-cell precursors in scid mice: Normal numbers of cells transformable with abelson murine leukemia virus (A-MuLV) von Fulop, G.M., Bosma, G.C., Bosma, M.J., Phillips, R.A.

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Murine lymphocytes with natural killer activity express CTL-derived serine protease genes von ANDERSON, S. K, HONG, K. K. Y, HOSKIN, D. W, ABRAMOW-NEWERLY, W, FULOP, G. M, PHILLIS, R. A, HOZUMI, N, RODER, J. C

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Salsolinol is a Putative Endogenous Neuro-intermediate Lobe Prolactin-Releasing Factor von Tóth, B. E., Homicskó, K., Radnai, B., Maruyama, W., DeMaria, J. E., Vecsernyés, M., Fekete, M. I. K., Fülöp, F., Naoi, M., Freeman, M. E., Nagy, G. M.

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Stress- as Well as Suckling-Induced Prolactin Release is Blocked by a Structural Analogue of the Putative Hypophysiotrophic Prolactin-Releasing Factor, Salsolinol von Bodnár, I., Mravec, B., Kubovcakova, L., Tóth, E. B., Fülöp, F., Fekete, M. I. K., Kvetnansky, R., Nagy, G. M.

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Salsolinol induces a decrease in cyclic AMP at the median eminence and an increase at the adenohypophysis in lactating rats von Radnai, Balázs, Kandár, Zoltán, Somogyvári-Vigh, Anikó, Mergl, Zsuzsanna, Oláh, Márk, Fülöp, Ferenc, Vecsernyés, Miklós, Nagy, György M.

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Binding site of salsolinol: its properties in different regions of the brain and the pituitary gland of the rat von Homicskó, Krisztián Gy, Kertész, István, Radnai, Balázs, Tóth, Béla E, Tóth, Géza, Fülöp, Ferenc, Fekete, Márton I.K, Nagy, György M

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Pivotal Role of an Endogenous Tetrahydroisoquinoline, Salsolinol, in Stress- and Suckling-Induced Release of Prolactin von RADNAI, B, MRAVEC, B, BODNÁR, I, KUBOVCAKOVA, L, FÜLÖP, F, FEKETE, M I K, NAGY, G M, KVETNANSKY, R

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Physiological role of salsolinol: Its hypophysiotrophic function in the regulation of pituitary prolactin secretion von Tóth, Béla E., Bodnár, Ibolya, Homicskó, Krisztián G., Fülöp, Ferenc, Fekete, Márton I.K., Nagy, György M.

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