Persistence of Contact Lens-Induced Corneal Parainflammation Following Lens Removal von Datta, Ananya, Lee, Ji Hyun, Truong, Tiffany, Flandrin, Orneika, Yang, Yujia, Evans, David J, Fleiszig, Suzanne M J

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TRPA1 and TPRV1 Ion Channels Are Required for Contact Lens-Induced Corneal Parainflammation and Can Modulate Levels of Resident Corneal Immune Cells von Datta, Ananya, Lee, Ji Hyun, Flandrin, Orneika, Horneman, Hart, Lee, Justin, Metruccio, Matteo M. E., Bautista, Diana, Evans, David J., Fleiszig, Suzanne M. J.

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Nerve‐associated transient receptor potential ion channels can contribute to intrinsic resistance to bacterial adhesion in vivo von Wan, Stephanie J., Datta, Ananya, Flandrin, Orneika, Metruccio, Matteo M. E., Ma, Sophia, Nieto, Vincent, Kroken, Abby R., Hill, Rose Z., Bautista, Diana M., Evans, David J., Fleiszig, Suzanne M. J.

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Contact lens-induced corneal parainflammation involving Ly6G+ cell infiltration requires IL-17A and γδ T cells von Datta, Ananya, Truong, Tiffany, Lee, Ji Hyun, Horneman, Hart, Flandrin, Orneika, Lee, Justin, Kumar, Naren G., Caspi, Rachel R., Evans, David J., Fleiszig, Suzanne M.J.

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Autonomous regulation of retinal insulin biosynthesis in diabetes von Jones, Malita A., Jadeja, Ravirajsinh N., Flandrin, Orneika, Abdelrahman, Ammar A., Thounojam, Menaka C., Thomas, Shakera, Dai, Caihong, Xiao, Haiyan, Chen, Jian-Kang, Smith, Sylvia B., Bartoli, Manuela, Martin, Pamela M., Powell, Folami L.

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