Functional Prostacyclin Synthase Promoter Polymorphisms: Impact in Pulmonary Arterial Hypertension von STEARMAN, Robert S, CORNELIUS, Amber R, COGAN, Joy D, PHILLIPS, John A, TAYLOR, Matthew R, GRAHAM, Brian B, TUDER, Rubin M, LOYD, James E, GERACI, Mark W, XIAO LU, CONKLIN, David S, DEL ROSARIO, Mark J, LOWE, Anita M, ELOS, Mihret T, FETTIG, Lynsey M, WONG, Randall E, HARA, Naoko

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Combined deletion of Xrcc4 and Trp53 in mouse germinal center B cells leads to novel B cell lymphomas with clonal heterogeneity von Chen, Zhangguo, Elos, Mihret T, Viboolsittiseri, Sawanee S, Gowan, Katherine, Leach, Sonia M, Rice, Michael, Eder, Maxwell D, Jones, Kenneth, Wang, Jing H

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Mutations in the accessory subunit NDUFB10 result in isolated complex I deficiency and illustrate the critical role of intermembrane space import for complex I holoenzyme assembly von Friederich, Marisa W, Erdogan, Alican J, Coughlin, 2nd, Curtis R, Elos, Mihret T, Jiang, Hua, O'Rourke, Courtney P, Lovell, Mark A, Wartchow, Eric, Gowan, Katherine, Chatfield, Kathryn C, Chick, Wallace S, Spector, Elaine B, Van Hove, Johan L K, Riemer, Jan

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Interplay between Target Sequences and Repair Pathways Determines Distinct Outcomes of AID-Initiated Lesions von Chen, Zhangguo, Eder, Maxwell D, Elos, Mihret T, Viboolsittiseri, Sawanee S, Chen, Xiaomi, Wang, Jing H

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AID-initiated DNA lesions are differentially processed in distinct B cell populations von Chen, Zhangguo, Ranganath, Sheila, Viboolsittiseri, Sawanee S, Eder, Maxwell D, Chen, Xiaomi, Elos, Mihret T, Yuan, Shunzong, Yuan, Shunzhong, Hansen, Erica, Wang, Jing H

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Correction: AID-Initiated DNA Lesions Are Differentially Processed in Distinct B Cell Populations von Chen, Zhangguo, Ranganath, Sheila, Viboolsittiseri, Sawanee S, Eder, Maxwell D, Chen, Xiaomi, Elos, Mihret T, Yuan, Shunzong, Hansen, Erica, Wang, Jing H

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AID-Initiated DNA Lesions Are Differentially Processed in Distinct B cell Populations1 von Chen, Zhangguo, Ranganath, Sheila, Viboolsittiseri, Sawanee S., Eder, Maxwell D., Chen, Xiaomi, Elos, Mihret T., Yuan, Shunzhong, Hansen, Erica, Wang, Jing H.

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The innate immune system stimulating cytokine GM-CSF improves learning/memory and interneuron and astrocyte brain pathology in Dp16 Down syndrome mice and improves learning/memory... von Ahmed, Md. Mahiuddin, Wang, Athena Ching-Jung, Elos, Mihret, Chial, Heidi J., Sillau, Stefan, Solano, D. Adriana, Coughlan, Christina, Aghili, Leila, Anton, Paige, Markham, Neil, Adame, Vanesa, Gardiner, Katheleen J., Boyd, Timothy D., Potter, Huntington

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Chromosome Instability and Mosaic Aneuploidy in Neurodegenerative and Neurodevelopmental Disorders von Potter, Huntington, Chial, Heidi J, Caneus, Julbert, Elos, Mihret, Elder, Nina, Borysov, Sergiy, Granic, Antoneta

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ELEVATED LEVELS OF MOSAIC ANEUPLOIDY IN HUNTINGTON’S DISEASE von Caneus, Julbert, Elos, Mihret, Potter, Huntington

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P1‐213: ELEVATED LEVELS OF MOSAIC ANEUPLOIDY IN HUNTINGTON'S DISEASE von Caneus, Julbert, Elos, Mihret, Potter, Huntington

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GM‐CSF Treatment Increases the Number of Purkinje Cells in a Mouse Model of Down Syndrome von Ahmed, Md. Mahiuddin, Aghili, Leila, Wang, Athena Ching‐Jung, Elos, Mihret, Boyd, Timothy D., Chial, Heidi J, Potter, Huntington

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Elevated levels of mosaic aneuploidy in brain cells and fibroblast cell lines from human Huntington’s disease donors and in brain cells from Huntington’s disease mouse models von Elos, Mihret, Caneus, Julbert, Ahmed, Md. Mahiuddin, Grissom, Paula M., Markham, Neil, Johnson, Noah R, Chial, Heidi J, Potter, Huntington

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Role of mosaic aneuploidy in the development and progression of Huntington's disease: Developing topics von Elos, Mihret, Caneus, Julbert, Ahmed, Md Mahiuddin, Markham, Neil, Chial, Heidi J, Potter, Huntington

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Role of mosaic aneuploidy in the development and progression of Huntington's disease von Elos, Mihret, Caneus, Julbert, Ahmed, Md Mahiuddin, Markham, Neil, Chial, Heidi J, Potter, Huntington

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GM-CSF REVERSES MEMORY DEFICITS IN THE DP16 MOUSE MODEL OF DOWN SYNDROME von Ahmed, Md. Mahiuddin, Jung Wang, Athena Ching, Boyd, Timothy, Elos, Mihret, Gardiner, Katheleen J., Potter, Huntington

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P2‐162: GM‐CSF REVERSES MEMORY DEFICITS IN THE DP16 MOUSE MODEL OF DOWN SYNDROME von Ahmed, Md. Mahiuddin, Jung Wang, Athena Ching, Boyd, Timothy, Elos, Mihret, Gardiner, Katheleen J., Potter, Huntington

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P4‐032: GM‐CSF REVERSES MEMORY DEFICITS IN NORMAL AGED MICE AND IN THE DP(16)1YEY MOUSE MODEL OF DOWN SYNDROME von Ahmed, Md. Mahiuddin, Jung Wang, Athena Ching, Boyd, Timothy, Elos, Mihret, Chial, Heidi J., Gardiner, Katheleen J., Potter, Huntington

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GM-CSF REVERSES MEMORY DEFICITS IN NORMAL AGED MICE AND IN THE DP(16)1YEY MOUSE MODEL OF DOWN SYNDROME von Ahmed, Md. Mahiuddin, Jung Wang, Athena Ching, Boyd, Timothy, Elos, Mihret, Chial, Heidi J., Gardiner, Katheleen J., Potter, Huntington

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AID-initiated DNA lesions are differentially processed in distinct B cell populations (IRM8P.701) von Wang, Jing, Chen, Zhangguo, Ranganath, Sheila, Viboolsittiseri, Sawanee, Eder, Maxwell, Elos, Mihret, Chen, Xiaomi, Hansen, Erica

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