The thrombopoietin receptor P106L mutation functionally separates receptor signaling activity from thrombopoietin homeostasis von Stockklausner, Clemens, Klotter, Anne-Christine, Dickemann, Nicole, Kuhlee, Isabelle N., Duffert, Christin M., Kerber, Carolin, Gehring, Niels H., Kulozik, Andreas E.

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The Predominant Activation of Alternative Signaling Pathways Explains the Differential Propensity of Different c-Mpl Gain-of-Function Mutations to Promote Myeloproliferative Malign... von Stockklausner, Clemens, Duffert, Christin Maria, Dickemann, Nicole, Klotter, Anne Christine, Kuhlee, Isabelle Nadine, Kerber, Carolin, Kulozik, Andreas E.

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