The non-dystrophic myotonias: molecular pathogenesis, diagnosis and treatment von Matthews, E., Fialho, D., Tan, S. V., Venance, S. L., Cannon, S. C., Sternberg, D., Fontaine, B., Amato, A. A., Barohn, R. J., Griggs, R. C., Hanna, M. G.

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The primary periodic paralyses: diagnosis, pathogenesis and treatment von Venance, S. L., Cannon, S. C., Fialho, D., Fontaine, B., Hanna, M. G., Ptacek, L. J., Tristani-Firouzi, M., Tawil, R., Griggs, R. C.

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Mexiletine block of disease-associated mutations in S6 segments of the human skeletal muscle Na+ channel von Takahashi, M P, Cannon, S C

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Spectrum of sodium channel disturbances in the nondystrophic myotonias and periodic paralyses von Cannon, Stephen C.

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The Human Skeletal Muscle Na Channel Mutation R669H Associated with Hypokalemic Periodic Paralysis Enhances Slow Inactivation von Struyk, Arie F, Scoggan, Kylie A, Bulman, Dennis E, Cannon, Stephen C

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Sodium channel defects in myotonia and periodic paralysis von CANNON, S. C

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Defective slow inactivation of sodium channels contributes to familial periodic paralysis von HAYWARD, L. J, SANDOVAL, G. M, CANNON, S. C

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Ion-channel defects and aberrant excitability in myotonia and periodic paralysis von Cannon, Stephen C

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The common missense mutation D489N in TRIM32 causing limb girdle muscular dystrophy 2H leads to loss of the mutated protein in knock-in mice resulting in a Trim32-null phenotype von Kudryashova, Elena, Struyk, Arie, Mokhonova, Ekaterina, Cannon, Stephen C., Spencer, Melissa J.

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Loss of the neural integrator of the oculomotor system from brain stem lesions in monkey von Cannon, S. C, Robinson, D. A

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A new mutation in a family with cold-aggravated myotonia disrupts Na+ channel inactivation von WU, F.-F, TAKAHASHI, M. P, PEGORARO, E, ANGELINI, C, COLLESELLI, P, CANNON, S. C, HOFFMAN, E. P

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Theoretical reconstruction of myotonia and paralysis caused by incomplete inactivation of sodium channels von Cannon, S.C., Brown, R.H., Corey, D.P.

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Functional expression of sodium channel mutations identified in families with periodic paralysis von Cannon, Stephen C., Strittmatter, Stephen M.

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Myasthenic Syndrome Caused by Mutation of the SCN4A Sodium Channel von Tsujino, Akira, Maertens, Chantal, Ohno, Kinji, Shen, Xin-Ming, Fukuda, Taku, Harper, C. Michael, Cannon, Stephen C., Engel, Andrew G.

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Inactivation defects caused by myotonia-associated mutations in the sodium channel III-IV linker von Hayward, L J, Brown, Jr, R H, Cannon, S C

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Loss of Na+ channel inactivation by anemone toxin (ATX II) mimics the myotonic state in hyperkalaemic periodic paralysis von Cannon, S C, Corey, D P

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MOD-1 is a serotonin-gated chloride channel that modulates locomotory behaviour in C. elegans von Ranganathan, Rajesh, Horvitz, H. Robert, Cannon, Stephen C

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GAP-43 Augments G Protein-Coupled Receptor Transduction in Xenopus laevis Oocytes von Strittmatter, Stephen M., Cannon, Stephen C., Ross, Elliott M., Higashijima, Tsutomu, Fishman, Mark C.

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Modification of the Na+ current conducted by the rat skeletal muscle α subunit by coexpression with a human brain β subunit von CANNON, S. C, MCCLATCHEY, A. I, GUSELLA, J. F

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Sodium channel inactivation is impaired in equine hyperkalemic periodic paralysis von Cannon, S. C, Hayward, L. J, Beech, J, Brown, R. H., Jr

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