Na+/Ca2+ exchange and Na+/K+‐ATPase in the heart von Shattock, Michael J., Ottolia, Michela, Bers, Donald M., Blaustein, Mordecai P., Boguslavskyi, Andrii, Bossuyt, Julie, Bridge, John H. B., Chen‐Izu, Ye, Clancy, Colleen E., Edwards, Andrew, Goldhaber, Joshua, Kaplan, Jack, Lingrel, Jerry B., Pavlovic, Davor, Philipson, Kenneth, Sipido, Karin R., Xie, Zi‐Jian

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Activation of reverse Na+–Ca2+ exchange by the Na+ current augments the cardiac Ca2+ transient: evidence from NCX knockout mice von Larbig, Robert, Torres, Natalia, Bridge, John H. B., Goldhaber, Joshua I., Philipson, Kenneth D.

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Novel Features of the Rabbit Transverse Tubular System Revealed by Quantitative Analysis of Three-Dimensional Reconstructions from Confocal Images von Savio-Galimberti, Eleonora, Frank, Joy, Inoue, Masashi, Goldhaber, Joshua I., Cannell, Mark B., Bridge, John H.B., Sachse, Frank B.

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Strain Transfer in Ventricular Cardiomyocytes to Their Transverse Tubular System Revealed by Scanning Confocal Microscopy von McNary, Thomas G., Bridge, John H.B., Sachse, Frank B.

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Na/Ca exchange and contraction of the heart von Ottolia, Michela, Torres, Natalia, Bridge, John H.B, Philipson, Kenneth D, Goldhaber, Joshua I

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Mechanisms regulating Ca2+ release in cardiomyocytes von Bridge, John H B, Torres, Natalia S

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Sodium-Calcium Exchange Is Essential for Effective Triggering of Calcium Release in Mouse Heart von Neco, Patricia, Rose, Beth, Huynh, Nhi, Zhang, Rui, Bridge, John H.B., Philipson, Kenneth D., Goldhaber, Joshua I.

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Cardiac Resynchronization Therapy Reduces Subcellular Heterogeneity of Ryanodine Receptors, T-Tubules, and Ca2+ Sparks Produced by Dyssynchronous Heart Failure von Li, Hui, Lichter, Justin G, Seidel, Thomas, Tomaselli, Gordon F, Bridge, John H B, Sachse, Frank B

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Effect of Metabolic Inhibition on Couplon Behavior in Rabbit Ventricular Myocytes von Chantawansri, Chana, Huynh, Nhi, Yamanaka, Jun, Garfinkel, Alan, Lamp, Scott T., Inoue, Masashi, Bridge, John H.B., Goldhaber, Joshua I.

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Na+ currents are required for efficient excitation–contraction coupling in rabbit ventricular myocytes: a possible contribution of neuronal Na+ channels von Torres, Natalia S., Larbig, Robert, Rock, Alex, Goldhaber, Joshua I., Bridge, John H. B.

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Neuronal Na+ channel blockade suppresses arrhythmogenic diastolic Ca2+ release von Radwański, Przemysław B, Brunello, Lucia, Veeraraghavan, Rengasayee, Ho, Hsiang-Ting, Lou, Qing, Makara, Michael A, Belevych, Andriy E, Anghelescu, Mircea, Priori, Silvia G, Volpe, Pompeo, Hund, Thomas J, Janssen, Paul M L, Mohler, Peter J, Bridge, John H B, Poelzing, Steven, Györke, Sándor

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Revealing the Cellular Basis of Heart Failure von Bridge, John H.B., Savio, Eleonora

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Quantitative Analysis of Cardiac Tissue Including Fibroblasts Using Three-Dimensional Confocal Microscopy and Image Reconstruction: Towards a Basis for Electrophysiological Modelin... von Schwab, B. C., Seemann, G., Lasher, R. A., Torres, N. S., Wulfers, E. M., Arp, M., Carruth, E. D., Bridge, J. H. B., Sachse, F. B.

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Metabolic Inhibition Alters Subcellular Calcium Release Patterns in Rat Ventricular Myocytes: Implications for Defective Excitation-Contraction Coupling During Cardiac Ischemia and... von Fukumoto, Gary H, Lamp, Scott T, Motter, Christi, Bridge, John H.B, Garfinkel, Alan, Goldhaber, Joshua I

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Mechanical modulation of the transverse tubular system of ventricular cardiomyocytes von McNary, Thomas G., Spitzer, Kenneth W., Holloway, Hilary, Bridge, John H.B., Kohl, Peter, Sachse, Frank B.

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Ca2+ Sparks in Rabbit Ventricular Myocytes Evoked by Action Potentials: Involvement of Clusters of L-Type Ca2+ Channels von Inoue, Masashi, Bridge, John H.B

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Allosteric Activation of Na+-Ca2+ Exchange by L-Type Ca2+ Current Augments the Trigger Flux for SR Ca2+ Release in Ventricular Myocytes von Sobie, Eric A., Cannell, Mark B., Bridge, John H.B.

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Dyssynchronous Ca2+ Sparks in Myocytes From Infarcted Hearts von Litwin, Sheldon E, Zhang, Dongfang, Bridge, John H.B

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Properties of Ca2+ sparks evoked by action potentials in mouse ventricular myocytes von Bridge, John H. B., Ershler, Philip R., Cannell, Mark B.

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What Are the Consequences of Phosphorylation and Hyperphosphorylation of Ryanodine Receptors in Normal and Failing Heart? von Bridge, John H.B, Savio-Galimberti, Eleonora

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