IL-1β, IL-23, and TGF-β drive plasticity of human ILC2s towards IL-17-producing ILCs in nasal inflammation von Golebski, Korneliusz, Ros, Xavier R., Nagasawa, Maho, van Tol, Sophie, Heesters, Balthasar A., Aglmous, Hajar, Kradolfer, Chantal M. A., Shikhagaie, Medya M., Seys, Sven, Hellings, P. W., van Drunen, Cornelis M., Fokkens, Wytske J., Spits, Hergen, Bal, Suzanne M.

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Hobit and Blimp‐1 regulate TRM abundance after LCMV infection by suppressing tissue exit pathways of TRMprecursors von Parga‐Vidal, Loreto, Taggenbrock, Renske L.R.E., Beumer‐Chuwonpad, Ammarina, Aglmous, Hajar, Kragten, Natasja A.M., Behr, Felix M., Bovens, Astrid A., van Lier, Rene A.W., Stark, Regina, van Gisbergen, Klaas P.J.M.

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Hobit and Blimp-1 regulate T RM abundance after LCMV infection by suppressing tissue exit pathways of T RM precursors von Parga-Vidal, Loreto, Taggenbrock, Renske L R E, Beumer-Chuwonpad, Ammarina, Aglmous, Hajar, Kragten, Natasja A M, Behr, Felix M, Bovens, Astrid A, van Lier, Rene A W, Stark, Regina, van Gisbergen, Klaas P J M

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IL-1β, IL-23, and TGF-β drive plasticity of human ILC2s towards IL-17-producing ILCs in nasal inflammation von Golebski, Korneliusz, Ros, Xavier R, Nagasawa, Maho, van Tol, Sophie, Heesters, Balthasar A, Aglmous, Hajar, Kradolfer, Chantal M.A, Shikhagaie, Medya M, Seys, Sven, Hellings, P.W, van Drunen, Cornelis M, Fokkens, Wytske J, Spits, Hergen, Bal, Suzanne M

Volltext

Hobit and Blimp-1 regulate TRM abundance after LCMV infection by suppressing tissue exit pathways of TRM precursors von Parga-Vidal, Loreto, Taggenbrock, Renske L R E, Beumer-Chuwonpad, Ammarina, Aglmous, Hajar, Kragten, Natasja A M, Behr, Felix M, Bovens, Astrid A, van Lier, Rene A W, Stark, Regina, van Gisbergen, Klaas P J M

Volltext