Heterozygous Peripheral Myelin Protein 22-Deficient Mice Are Affected by a Progressive Demyelinating Tomaculous Neuropathy von Adlkofer, Katrin, Frei, Regula, Neuberg, Dirk H.-H, Zielasek, Jurgen, Toyka, Klaus V, Suter, Ueli

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Hypermyelination and demyelinating peripheral neuropathy in Pmp22-deficient mice von ADLKOFER, K, MARTINI, R, AGUZZI, A, ZIELASEK, J, TOYKA, K. V, SUTER, U

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Hypermyelination and demyelinating peripheral neuropathy in Pmp22 -deficient mice von Adlkofer, Katrin, Martini, Rudolf, Aguzzi, Adriano, Zielasek, Jürgen, Toyka, Klaus V, Suter, Ueli

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Aberrant Protein Trafficking inTremblerSuggests a Disease Mechanism for Hereditary Human Peripheral Neuropathies von Naef, Roland, Adlkofer, Katrin, Lescher, Birgit, Suter, Ueli

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Impaired Differentiation of Schwann Cells in Transgenic Mice with Increased PMP22 Gene Dosage von Magyar, Josef P, Martini, Rudolf, Ruelicke, Thomas, Aguzzi, Adriano, Adlkofer, Katrin, Dembic, Zlatko, Zielasek, Jurgen, Toyka, Klaus V, Suter, Ueli

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Aberrant protein trafficking in Trembler suggests a disease mechanism for hereditary human peripheral neuropathies von Naef, R, Adlkofer, K, Lescher, B, Suter, U

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Analysis of compound heterozygous mice reveals that the Trembler mutation can behave as a gain-of-function allele von Adlkofer, Katrin, Naef, Roland, Suter, Ueli

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Analysis of compound heterozygous mice reveals that theTrembler mutation can behave as a gain-of-function allele von Adlkofer, Katrin, Naef, Roland, Suter, Ueli

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Hereditary neuromyotonia: a mouse model associated with deficiency or increased gene dosage of the PMP22 gene von TOYKA, KLAUS V, ZIELASEK, JÜRGEN, RICKER, KENNETH, ADLKOFER, KATRIN, SUTER, UELI

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